연구 그림 — WeightCited

Figure 3 Diagram

Pathophysiological mechanisms shared between osteoporosis and sarcopenia are diagrammed, including hormonal, nutritional, and mechanical loading factors.

Osteosarcopenia: epidemiology, diagnosis, and treatment-facts and numbers.

Figure 1. Likely mechanisms through which dietary fiber affects the functions of the gastrointestinal tract. Dietary fiber acts as a prebiotic to intestinal microbiota that causes changes in their composition and induces the growth of beneficial bacteria.

Figure 3 Diagram

Dietary fiber acts as a prebiotic for intestinal microbiota, promoting growth of beneficial bacteria and production of short-chain fatty acids. This diagram illustrates multiple mechanisms through which fiber affects gastrointestinal function, including modulation of gut microbiome composition and intestinal barrier integrity.

Dietary fiber in irritable bowel syndrome (Review).

Figure 6 Diagram

Neurological consequences of chronic manganese exposure are summarized, including cognitive impairment, motor dysfunction, and emotional disturbances. The symptoms collectively termed manganism share clinical features with Parkinson's disease.

Consequences of Disturbing Manganese Homeostasis.

Figure 7 Diagram

Manganese's dual role as both essential nutrient and potential toxin is explored through its effects on enzymatic systems. Superoxide dismutase, arginase, and glutamine synthetase all require manganese as a cofactor for normal function.

Consequences of Disturbing Manganese Homeostasis.

Figure 8 Diagram

A comprehensive diagram maps the systemic effects of manganese dysregulation across multiple organ systems. Both deficiency and excess disrupt metabolic processes, with the nervous system being particularly vulnerable to manganese imbalance.

Consequences of Disturbing Manganese Homeostasis.

Figure 4. A model of the imbalance in the Mn transport system.

Figure 10 Diagram

A model of manganese transport imbalance depicts the interplay between uptake, distribution, and excretion mechanisms. Disruption of transporters such as SLC30A10 and SLC39A14 can lead to pathological manganese accumulation in target tissues.

Consequences of Disturbing Manganese Homeostasis.

Figure 8 Diagram

A vegan food pyramid illustrates recommended dietary proportions for plant-based nutrition, organizing food groups by suggested intake frequency to support metabolic health.

Vegan Diet Health Benefits in Metabolic Syndrome.

Figure 6. Effects of vegan diet on metabolic syndrome. APO, apolipoprotein; FM, fat mass; SFAs, saturated fatty acids; ↑ increase; ↓ decrease.

Figure 11 Diagram

Beneficial effects of a vegan diet on metabolic syndrome components are mapped, showing improvements in apolipoprotein profiles, fat mass reduction, and decreased saturated fatty acid intake alongside their downstream metabolic consequences.

Vegan Diet Health Benefits in Metabolic Syndrome.

Figure 1. The iRISA syndrome [76] in addiction is centrally controlled by dopamine in the brain, while asynchronization, presumed to be linked to cue sensitivity in digital addiction [50–54], is centrally controlled by serotonin. A deﬁcit in both neurotra

Figure 6 Diagram

The impaired Response Inhibition and Salience Attribution (iRISA) syndrome model is illustrated, showing how dopamine-mediated reward pathways in the brain drive cue sensitivity in digital addiction and contribute to sleep disruption.

Digital Addiction and Sleep.

Figure 7 Diagram

Neurobiological mechanisms linking excessive digital device use to disrupted sleep architecture are outlined, connecting screen-mediated blue light exposure and dopaminergic reward activation to circadian rhythm disturbances.

Digital Addiction and Sleep.

Figure 2 Diagram

Approaches for managing antibiotic-associated dysbiosis are summarized, including strategies to restore gut microbial diversity after antibiotic treatment. The figure outlines interventions such as probiotics and microbiota-targeted therapies that may help mitigate dysbiosis in populations predisposed to gut disruption.

Current understanding of antibiotic-associated dysbiosis and approaches for its management.

Fig. 1. The underlying mechanism of depression. Brain insulin resistance develops due to the failure of brain cells to respond to insulin activity. The hippocampus, hypothalamus, and cortex regions of the central nervous system regulate insulin levels in

Figure 3 Diagram

Brain insulin resistance mechanisms underlying depression are depicted, showing how HPA axis dysregulation, reduced anterior cingulate cortex volume, and impaired hippocampal function contribute to the condition.